Our main office is in Bethesda, Bldg 37/Rm 3041. Â Office hours for drop-ins are from 10 am -12 noon on Fridays.
Requests for any type of Bioinformatics support or questions about Pipeliner should be filed through the CCBR Project Submission Form.
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Upcoming Classes
Publications
- 06/21/2022 - Host CLIC4 expression in the tumor microenvironment is essential for breast cancer metastatic competence
- 05/09/2022 - A phenotypic signature that identifies neoantigen-reactive T cells in fresh human lung cancers
- 03/01/2022 - A phase 2 study of Pembrolizumab in combination with Capecitabine and Oxaliplatin with molecular profiling in advanced biliary tract carcinoma
- 03/01/2022 - Remodeling the tumor microenvironment via blockade of LAIR-1 and TGF-? signaling enables PD-L1-mediated tumor eradication
- 02/11/2022 - Reusable single cell for iterative epigenomic analyses
- 02/01/2022 - Urine single-cell RNA sequencing in focal segmental glomerulosclerosis reveals inflammatory signatures
- 02/01/2022 - H3K27ac nucleosomes facilitate HMGN localization at regulatory sites to modulate chromatin binding of transcription factors
- 12/14/2021 - TNF plays a crucial role in inflammation by signaling via T cell TNFR2
- 12/01/2021 - Identifying and characterizing virus-encoded circular RNAs
- 12/01/2021 - The butyrophilin 1a1 knockout mouse revisited: Ablation of Btn1a1 leads to concurrent cell death and renewal in the mammary epithelium during lactation
- 11/17/2021 - Protein-RNA interactome analysis reveals wide association of KSHV ORF57 with host non-coding RNAs and polysomes
- 11/01/2021 - PERK signaling through C/EBP? contributes to ER stress-induced expression of immunomodulatory and tumor promoting chemokines by cancer cells
- 11/01/2021 - TP53 loss initiates chromosomal instability in fallopian tube epithelial cells
- 10/20/2021 - Ecdysoneless protein regulates viral and cellular mRNA splicing to promote cervical oncogenesis
- 10/07/2021 - CTCF is a barrier for totipotent 2C-like reprogramming
- 10/01/2021 - The ETS transcription factor ERF controls the exit from the naïve pluripotent state in a MAPK-dependent manner